Pathophysiology Of Hcvd, Dm2, Cvd (left Basal Ganglia)

PREDISPOSING Non-Modifiable AGE (loss of blood vessel elasticity) GENDER FAMILY (hereditary) Modifiable LIFESTYLE DIET (high fat, sweet)

Insufficient insulin secretion/ production of the pancreas Glucose is not metabolize by the body

PRECIPITATING Diabetes Mellitus

Accumulation of glucose in the blood stream Increase fat deposit on the walls of the blood vessels Narrowing of blood vessel (coronary artery)

Increase viscosity of blood/ sluggish flow of blood Evaluate level of blood glucose/ increase concentration of sugar in blood

Atherosclesrosis

Pressure on the wall of blood

Decrease blood flow through the coronary S/sx: BP: 140/90 mmHg

Increase Peripheral Vascular resistance Some fragments joins in circulation (EMBOLISM)

CORONARY ARTERY

Goes to small vessels and clog (deep penetrating arteries especially lacunar region)

Decrease blood flow to the heart Increase blood pressure

S/sx: CBG monitoring: 103166mg/dL

Obstruction/ interruption of O2 supply due to decreased blood supply

HCVD Lacunar infarct Severe elevation in BP w/o progressive target Hypertensiv e

INFARCTION

Prognosis

If treated: • Nursing Management • Diet Management • Pharmacologic Medication: (Aspirin, ACE inhibitor, Nitroglycerin for angina) • Oxygen Recove ry

Decrease oxygen supply to basal ganglia (L)

If untreated: Further complication can occur: • Ischemia • Angina pectoris • MI • Heart attack • CVA

Death

Influences autonomic nervous Responsible for cerebellar function Also affects motor skills of chewing & swallowing (DYSPHAGIA) Difficulty in chewing & swallowing could lead to decrease appetite Causing weight loss