Pathophysiology of Congestive heart failure
MODIFIABLE RISK FACTORS
Hypertension Coronary Heart disease Valvular disease Arteriosclerosis Atherosclerosis Diabetes Obesity Viruses Diet Heart attack Smoking Congenital heart defects
Change in Endothelial tissue Weakening of right ventricle
Right ventricular hypertrophy
Non modifiable risk factors
Narrowing of vessel with plaque Thrombus formation
Age (60-69) Gender Ethnicity (south asians in UK are twice likely to develop CAD; African Caribbean have increased average risk)
Increase workload; walls thicken Backflow of blood circulation Anaerobic metabolism
Myocardial Dysfunction Right sided heart failure Myocardial Failiure
Decrease systemic bloodflow
Altered systemic perfusion
Anterior Myocardial Infarction
Lactic acid build up
ST Depression Chest pain Troponin 1
Neurohormonal activation
Activation of RAAS
Vasoconstriction of small arterioles
Increase sympathetic activity
Release of epinephrine and norepinephrine
Increase myocardial workload
Increase peripheral vascular resistance
Increase aldosterone release
UNDERLINED- Risk Factors of Patient RED- Symptoms Manifested by Patient GREY- Complications
Atrial Fibrillation
Prolonged QT wave
Prolonged P wave
Heart Attack Decrease in perfusion in tissues DEATH
EDEMA
Decrease ventricular inotropy
Decreased Stroke volume
Increase sodium and water retention
Increase BUN
Decrease end systolic volume
Systemic Hypoxemia Systemic Hypoxia Use of Accessory muscles for Respiration
Fatigue