DEFINITION IT IS DEFINED AS A DISEASE USUALLY EITHER INFECTIOUS OR TOXIC IN NATURE CAUSED BY AGENTS THAT ENTER THE BODY THROUGH INGESTION OF FOOD
FOOD BORNE INFECTION & ITS DIET THERAPY BACTERIAL DISEASE
TYPHOID
DIARRHOEA
DYSENTRY
TYPHOID DEFINITION IT IS AN INFECTIOUS DISEASE CHARACTERISED BY DEFINITE LESIONS IN PAYERS PATCHES,MESENTRIC GLANDS &SPLEEN ACCOMPANIED BY FEVER,HEADACHE & ABDOMINAL PAIN
CAUSITIVE ORGANISM SALMONELLA TYPHI
MODE OF TRANSMISSION CONTAIMINATED WATER RARELY BY CONTACT IMPROPERLY COOKED STARCHY FOODS & PASTERY UNSAFE RAW FRUITS & VEGETABLES CONTAIMINATION BY HANDS SOMETIMES BY FLIES
INCUBATION PERIOD USUALLY ONE TO THREE WEEKS (DEPENDING UPON THE INFECTIVE DOSE)
SIGN &SYMPTOMS
FEVER(103 F TO 104 F) LOSS OF APPETITE WEAKNESS HEADACHE BRADYCARDIA MENTAL DULLNESS & SLIGHT DEAFNESS STOMACH PAIN
SPLEENOMEGALY DELIRIUM NOSE BLEED
TREATMENT
REST KEEPING THE PATIENT WARM ANTIBIOTIC THERAPY MODIFIED DIET IMMUNIZATION GENERAL CARE & ISOLATION
DISINFECTION OF ALL THE DISCHARGES PROPER STERELIZATION OF ALL THE ARTICLES CHRONIC CARRIERS SHOULD BE INVESTIGATED
DIETARY MANAGEMENT PRINCIPLES
TO MAINTAIN ADEQUATE NUTRITION TO RESTORE POSITIVE NITROGEN BALANCE TO PROVIDE RELIEF FROM SYMPTOMS
TO CORRECT & MAINTAIN WATER & ELECTROLYTE IMBALANCE
DIET PATTERN
HIGH CALORIE HIGH PROTIEN FULL FLUID DIET BLAND, LOW FIBRE , SOFT DIET SRONGLY FLAVOURED FOOD SHOULD BE AVOIDED WELL COOKED , WELL MASHED SEMISOLID FOOD CAN BE GIVEN SHOULD BE EASILY DIGESTIBLE
DIARRHOEA FREEQUENT PASSAGE OF THE LOOSE WATERY & UNFORMED STOOL WHICH MAY CONTAIN BLOOD OR MUCOUS
TYPES
ACUTE CHRONIC
ACUTE DIARRHOEA CHRACTERISED BY SUDDEN ONSET & FREEQUENT PASSAGE OF WATERY & UNFORMED STOOL
INCUBATION PERIOD IT LAST FOR ONLY 24-48 HOURS
ETIOLOGY CONTAIMINATED FOOD & WATER FOOD SENSITIVITY OR ALLERGY PROTIEN ENERGY MALNUTRITION VIT A DEFICIENCY CHOLERA TYPHOID OR INFECTIOUS TONSILITIS
ARSENIC LEAD & MERCURY POISIONING TENTION & ANXIETY
SYMPTOMS
PATIENT MAY PASS SEVERAL STOOL ABDOMINAL PAIN CRAMPS WEAKNESS FEVER SOMETIMES AND VOMITING
CHRONIC DIARRHOEA IT PERSIST FOR A LONGER TIME EVEN SEVERAL WEEKS & MAY PASS UNFORMED STOOL IN A DAY ETIOLOGY
DM CHRONIC ALCOHLISM CANCER OF COLON PROLONGED INTAKE OF LAXATIVE LIVER CIRRHOSIS
SYMPTOMS
NURITIONAL DEFICIENCY
DIETARY MANAGEMENT
DIET SHOULD BE EASILY DIGESTIBLE SOFT BLAND DIET LOW FIBRE & FAT WELL COOKED SOFT SEMISOLID FOOD DRINK PLENTY OF FLUIDS
CONTROL MEASURES ORS APPROPRIATE FEEDING APPROPRIATE DRUGS SHOULD BE GIVEN PROPER HEALTH EDUCATION
DYSENTRY SEVERE DIARRHOEA CALLED AS DYSENTRY CHARECTERISED BY
FREEQUENT PASSAGE OF SMALL WATERY STOOL OFTEN CONTAINING BLOOD & MUCOUS ACCOMPANIED BY SEVERE ABDOMINAL PAIN ULCERATION OF WALLS OF INTESTINE
ETIOLOGY
CAUSED BY ENTAAMOEBA HISTOLICA
MODE OF TRANSMISSION
CONTAIMINATED WATER SREAD FROM HAND TO MOUTH TRANSFER OF FRESH FEACES BY COTAIMINATED RAW VEGETABLES, BY FLIES,BY SOILED HANDS OF FOOD HANDLERS & BY COTAIMINATED WATER
INCUBATION PERIOD FROM FEW DAYS TO SEVERAL MONTH OR YEAR SIGN & SYMPTOMS
FREEQUENT PASSING STOOL CONTAINING BLOOD & MUCOUS FEVER WITH CHILLS ABDOMINAL PAIN MILD TO SEVERE DEGREE OF DEHYDRATION
CONTROL MEASURES MAINTAIN HYGIENE SANITARY DISPOSAL OF HUMAN FECES SAFE WATER SUPPLY FLY CONTROL MEASURES ISOLATION INVESTIGATION HEALTH EDUCATION
DIETARY MANAGEMENT SOFT BLAND DIET EASILY DIGESTABLE LOW IN FIBRE & FAT HIGH CALORIE HIGH CALCIUM & IRON RICH DIET WELL COOKED SEMISOLID FOOD HIGH INTAKE OF FLUID HIGH PROTIEN
VIRAL HEPATITISIS IS AN INFLAMMATION OF LIVER ETIOLOGY
FECAL CONTAIMINATION OF FOOD & WATER POOR HYGIENE & SANITATION CONTAIMINATED BLOOD PRODUCTS VIRUS POORELY STERELISED NEEDLE
CAUSITIVE AGENT
VIRUS
INCUBATION PERIOD
15-50DAYS
MODE OF TRANSMISSION
PERSON TO PERSON CONTACT FECAL ORAL ROUTE CONAIMINATED WATER & FOOD LIKE MILK SALADS BAKERYY PRODUCTS INFECTED BLOOD & BLOOD PRODUCTS NEEDLE PRIC OF INFECTED PERSON
SIGN & SYMPTOMS
LOSS OF APPITITE FATIGUE MALAISE GENERALISED ITCHING NAUSEA & VOMITING LOW GRADE FEVER PALE &CLAY COLOURED STOOL ABNORMAL TASTE ABDOMINAL PAIN INDIGESTION&TENDERNESS
TYPES
ACUTE CHRONIC
PREVENTION &CONTROL DIETARY MANAGEMENT
FOOD SHOULD BE EASILY DIGESTIBLE & SOFT IN MILD CASES NORMAL CONSISTENCY DIET,IN ACUTE LIQUID TO SOFT DIET,IN SEVERE TUBE FEEDING HIGH CALORIE (1500-2000 K CAL)
HIGH PROTIEN & LOW FAT CARBOHYDRATE ADEQUATE(300-400gm)
FOODS INCLUDED
SUGAR GLUCOSE HONEY CEREALS PULSES MILK & MILK PRODUCTS EGGS, FRUITS& VEGETABLES
FOODS EXCLUDED
FRIED & FATTY FOODS OILS NUTS & OILSEED STRONGLY FLAVOURED VEGETABLES INTAKE OF ALCOHOL IS RESTRICTED FOR A YEAR AFTER THE ATTACK
POLIOMYLITIS IT IS PRIMARILY AN INFECTIOUS DISEASE OF HUMAN ELIMENTARY TRACT BUT MAY EFFECT THE BRAIN,SPINAL CORD&NERVES RESULTING IN PARALYSIS
EPIDEMIOLOGICAL FACTOR
CAUSITIVE AGENT IS POLIOVIRUIS SOURCE OF INFECTION IS FAECES & PHARENGEAL SECREATION
AGE BETWEEN 1TO 2 YEARS ENVIRONMENTAL CONDITION PERIOD OF INFECTIVITY-7-10 DAYS BEFORE & AFTER ONSET OF SYMPTOMS TRANSMISSION-FAECAL ORAL ROUTE DROPLET INFECTION INCUBATION PERIOD- 7-21 DAYS
CLINICAL FEATURES
90% ASYMPTOMATIC FEVER HEADACHE DIARRHOEA VOMITING DROWSINESS PAIN WEAKNESS STIFFNESS OF NECK & BACK PARALYSIS OF EFFECTED LIMB
PREVENTION DIETARY MANAGEMENT
FULL NORMAL DIET HIGH FIBRE, HIGH CALORIE LOW FAT PLENTY OF FLUID PROTIEN,MINERAL & VITAMIN SHOULD BE ADEQUATE
AMOEBIASIS AGENT FACTOR CAUSED BY PATHOGENIC STRAINS OF HISTOLYTICA RESERVOIR OF INFECTION MAN IMMEDIATE SOURCE IS FEASES CARRIERS PERIOD OF COMMUNICABILITY MAY BE SEVERAL YEARS
HOST FACTOR CAN OCCUR AT ANY AGE NO SEX OR RACIAL DIFFERENCE ENVIRONMENTAL FACTOR RELATED TO POOR SANITATION& SOCIO ECONOMIC STATUS TO CLIMATE INCUBATION PERIOD 2 TO 4 WEEKS OR LONGER
MODE OF TRANSMISSION FEACAL ORAL ROUTE SEXUAL TRANSMISSION VECTOR PREVENTION & CONTROL DIETERY MANAGEMENT NORMAL DIET & EASILY DIGESTABLE SOFT BLAND DIET
HIGH IN FIBRE & ADEQUATE FAT PROTIEN & CARBOHYDRATE
ROUND WORM IT IS AN INTESTINAL INFESTATION . CAUSITIVE ORGANISM ASCARIS LUMBRICOIDES
MODE OF TRANAMISSION BY INGESTION OF INFECTED EGGS FROM SOIL CONTAIMINATED WITH HUMAN FEACES SALADS 7 OTHER FOOD EATEN RAW ARE THE COMMON VEHICLES BY CARRIER
INCUBATION PERIOD WORMS REACH MATURITY IN ABOUT 2 MONTHS AFTER THE EGGS ARE INGESTED BY MAN SIGN & SYMPTOMS IRREGULAR RESPIRATION SPASM OF COUGING FEVER ABDOMINAL PAIN VOMITING
RESTLESSNESS DISTURBED SLEEP PREVENTION & CONTROL DIETERY MANAGEMENT EASILY DIGESTIBLE , SOFT BLAND DIET WITH ADEQUATE FIBRE WELL COOKED SOFT & SEMISOLID FOOD
PLENTY OF FLUID LOW FAT HIGH VITAMINS & MINERAL RICH DIET
LATHYRISM IT IS A CRIPPING DISEASE CHARECTERISED BY PARALYSIS OF THE LEG MUSCLE OCCURING IN ADULTS CONSUMING LARGE QUANTITIES OF SEED OF L SATIVUS.
ETIOLOGY L SATIVUS SEED SIGN & SYMPTOMS STAGE FIRST – WEAKNESS OF THE LOWER LIMBS & SPASTICITY OF THE LEG MUSCLE STAGE SECONDFLEXION OF THE KNEES ARE MORE MARKED& INVERSION OF THE FOOT WITH A TENDENCY TO WALK ON TOES
STAGE THIRDSYMPTOMS ARE MORE MARKED SUBJECT CAN WALK ONLY WITH THE HELP OF CRUCHES OR STICK STAGE FOURTHKNEES BECOME COMPLETELY FLEXED & ERECT 7 WALKING BECOME IMPOSSIBLE & ATROPHY OF THIGH & LEFT MUSCLE
PREVENTION DIETERY MANAGEMENT CONSUMPTION OF L .SATIVUS SEED SHOULD BE AVOIDED
ENDEMIC ASCITIS PREVENTION
ERGOT IT IS A FIELD FUNGUS FOOD GRAINS LIKE SORGHUM , WHEAT, BAJRA , RYE HAVE A TENDENCY TO GET INFESTED BY THIS FUNGUS.
SIGN & SYMPTOMS NAUSEA VOMITING GIDDINESS
DROWSINESS PAINFUL CRAMPS IN LIMBS PREVENTION DIETERY MANAGEMENT SYMPTOMATIC DIET SHOULD BE GIVEN WITH AVOIDENCE OF ERGOT
ALATOXIN FUSARIUM TOXIN ASBESTOS NURSING MANAGEMENT SUMMARY & CONCLUSSION