CARDIOVASCULAR NCLEX REVIEW PART ONE 1- ANATOMY AND PHYSIOLOGY
Epidemiology
worldwide, millions of new cases of rheumatic fever are reported each year rheumatic fever follows a group A streptococcal infection prevention is simply to find and treat streptococcal pharyngitis with malnutrition and crowded living, rheumatic fever is most common in children between the ages of five and 15 rheumatic fever strikes most often during cool, damp weather. In the U.S., it is most common in the northern states no one knows how and why group A streptococcal infections cause the lesions called Aschoff bodies damage depends on site of infection: most often the mitral valve in females and the aortic valve in males malfunction of these valves leads to severe pericarditis, and sometimes pericardial effusion and fatal heart failure. Of those who survive this complication, about 20% die within ten years
Anatomy and Physiology Anatomy 1-Layers
Pericardium: fibrous sac that encloses the heart
Epicardium: covers exterior surface of heart muscle
Myocardium: muscular portion of the heart
Endocardium: lines cardiac chambers and covers surface of heart valves
Anatomy Continue 2-Chambers of heart ( illustration)
Right atrium: collecting chamber for incoming systemic venous system Right ventricle: propels blood into pulmonary system Left atrium: collects blood from pulmonary venous system Left ventricle: largest thick-walled muscle that acts as a highpressure pump which propels blood into the systemic arterial system
Anatomy Continue 3- Heart valves: membranous openings that allow one way blood flow Atrioventricular valves: prevent backflow from ventricles to atria during systole Tricuspid - valve between right atrium and right ventricle Mitral - valve between left atrium and left ventricle Semilunar valves prevent backflow from aorta and pulmonary arteries into ventricles during diastole Pulmonic - valve between the right ventricle and pulmonary artery Aortic - valve between left ventricle and aorta
Anatomy Continue 4- Blood supply to heart Arteries - coronary Right supplies right ventricle and the back part of the left ventricle Left supplies mostly left ventricle and septum Veins Coronary sinus - wide venous channel that drains five coronary veins into the right atrium Thebesian - the smallest coronary veins drain some venous blood directly into the right atrium and ventricle and the left ventricle
Anatomy Continue
Veins
Coronary sinus - wide venous channel that drains five coronary veins into the right atrium
The besian - the smallest coronary veins drain some venous blood directly into the right atrium and ventricle and the left ventricle
Anatomy Continue 5- Conduction system
SA (Sinoatrial) node -referred to as the pacemaker of the heart, and located in the right atrium Junctional tissue - often referred to as the atrioventricular node (AV node) Bundle branch Purkinje system - the electrical system located in the septum and into cardiac tissues
Physiology
Physiology 1-Function of the heart is the transport of oxygen, carbon dioxide, nutrients and waste products
Physiology Continue
2- Cardiac cycle consists of:
Systole - The phase of contraction during which the chambers eject blood Diastole - The phase of relaxation during which the chambers fill with blood When the heart pumps, myocardial layers contract and relax The atria and ventricles work in an asynchronous manner
Physiology Continue 3- Blood flow:
Deoxygenated blood enters the right atrium through the superior and inferior venae cavae This blood enters the right ventricle through the tricuspid valve Then the blood travels through the pulmonic valve to the pulmonary arteries and into the lungs Oxygenated blood returns from lungs through the pulmonary veins into the left atrium The blood then enters the left ventricle through the bicuspid (mitral) valve Finally, the blood, from the left ventricle, goes through the aortic valve into the aorta and
Physiology Continue:
4-
The heart itself is supplied with blood by the left and right coronary arteries, which are found at the base of the aorta above the aortic valves
Physiology Continue
The vascular system is a continuous network of blood vessels The arterial system consists of arteries, arterioles and capillaries and delivers oxygenated blood and nutrients to tissues Oxygen, carbon dioxide, nutrients, and metabolic waste are exchanged at the capillary level The venous system, veins and venules, returns the blood with carbon dioxide and metabolic wastes to the heart
Epidemiology
Epidemiology
May be acute or chronic
May occur at any age
Pericarditis may occur in up to 15% of persons with a transmural infarction.
Findings
Sharp chest pain often relieved by sitting upright and leaning forward
Pericardial friction rub
Dyspnea
Fever, sweating, chills
Dysrhythmias and EKG changes
Pulsus paradoxus
Client cannot lie flat without severe pain or dyspnea
DATA COLLECTION FOR CLIENTS WITH CARDIOVASCULAR DISORDERS
Diagnostics
Diagnostics History and physical exam Serum increased white blood cells sedimentation rate positive blood cultures if infection Antinuclar antibody (ANA) if due to connctive tissue disease EKG changes on 12-lead Echocardiography: to determine pericardial effusion or cardiac tamponade
Medical Management
Medical Management Antibiotics: to treat underlying infection Corticosteroids: if no response to NSAID or if effusion Anti inflammatory/analgesics: NSAID, ASA Avoid anticoagulants because they may increase the possibility of cardiac tamponade from bleeding risk Oxygen: to prevent tissue hypoxia Surgical Emergency pericardiocentesis if cardiac tamponade develops For recurrent constrictive pericarditis, partial pericardiectomy (pericardial window) or total pericardiectomy
Nursing interventions Manage pain and anxiety Semi-Fowler's or high-Fowler's position Mild analgesics to keep pain at 0 to 2; on a scale of 1 to 10 Medications to treat cause
The Cardio-Care Six The Cardio-Care Six THE CARDIO-CARE SIX: A,B,C,D,E,F
ADL: Help the client with activities of daily living and how to schedule activities that minimize cardiac stress.
Bed rest
Commode at bedside (it is less stressful to the heart than using a bedpan)
Diversions: offer diversions that don't stress the heart (e.g., no hand-held electronic games).
Elevate head of bed or sit client up to a position of comfort.
Feelings: plan time for the client to express his concerns.
Client and Family Teaching - Teach the Cardio Five
Maintain a pericardiocentesis set at the bedside in case of cardiac tamponade Assess respiratory, cardiovascular, and renal status q 1 to 2 hours in acute phase Observe for pericarditis complications dysrhythmias cardiac tamponade heart failure Observe for signs of infiltration or inflammation at the venipuncture site, a possible complication of long-term IV administration. Rotate the IV sites often.
TEACH THE CARDIO FIVE: TDDDS
Tests and treatments: discuss them in simple, culturally sensitive ways.
Drugs, their side effects, how long client will take them, and their expected effects.
Diet: balanced nutrition and restrictions (such as low sodium).
Disease, its management, when and what signs to report promptly: the 'watch-for s'.
Smoker? Stress benefits of stopping smoking, minimization of other stimulants - caffeine, chocolate, nonprescription drugs, herb cautions
Myocarditis
Myocarditis
Definition - An inflammatory condition of the myocardium
Epidemiology / Etiology May be acute or chronic and may occur at any age. Usually an acute virus and self-limited, but it may lead to acute heart failure. Etiologies: Viral infection Bacterial infection Fungal infection Serum sickness Rheumatic fever Chemical agent
As a complication of a collagen disease, i.e. SLE
Myocarditis
Findings Depends on the type of infection, degree of myocardial damage, capacity of myocardium to recover, and host resistance May be minor or unnoticed: fatigue and dyspnea, palpitations, occasional precordial discomfort manifested as a mild chest soreness and persistent fever Recent upper-respiratory infection with fever, viral pharyngitis, or tonsillitis Cardiac enlargement Abnormal heart sounds Possibly signs of heart failure such as pulsus alternans, dyspnea and crackles Tachycardia disproportionate to the degree of fever
Myocarditis
Myocarditis Nursing intervention
the cardio-care six with modified bedrest and less help with ADLs THE CARDIO-CARE SIX: A,B,C,D,E,F ADL: Help the client with activities of daily living and how to schedule activities that minimize cardiac stress. Bed rest Commode at bedside (it is less stressful to the heart than using a bedpan) Diversions: offer diversions that don't stress the heart (e.g., no hand-held electronic games). Elevate head of bed or sit client up to a position of comfort. Feelings: plan time for the client to express his concerns. assess for edema ; weigh daily; record intake and output (figure)
Myocarditis
assess for edema ; weigh daily; record intake and output monitor cardiovascular status watch for signs of leftsided heart failure (dyspnea, hypotension and tachycardia) check often for changes in cardiac rhythm or conduction monitor arterial blood gas levels as needed to ensure adequate oxygenation
Myocarditis
Endocarditis
Definition and related terms an infection of the endocardium, heart valves, or cardiac prosthesis resulting from bacterial or fungal invasion. endocarditis can be classified as native valve endocarditis endocarditis in I.V. drug users prosthetic valve endocarditis Epidemiology with proper treatment about 70% of clients recover. the prognosis is worse when endocarditis damages valves severely or involves a prosthetic valve infective endocarditis occurs in 50 to 60% of clients with previous valvular disorders. systemic lupus erythematosus (SLE) often leads to nonbacterial endocarditis. in 12% to 35% of clients with subacute endocarditis, lesions produce clots that show the findings of splenic, renal, cerebral or pulmonary infarction, or peripheral vascular occlusion.
Endocarditis
especially, a murmur that changes suddenly, or a new murmur that develops in the presence of a fever fever pericardial friction rub anorexia malaise clubbing of fingers petechiae of the skin, especially on the chest; conjunctiva, oral mucosa, abdomen splinter hemorrhage under the nails
Endocarditis
neurologic sequelae of embolus infarction of spleen: pain in the upper left quadrant of abdomen, radiating to the left shoulder, and abdominal rigidity infarction in kidney: hematuria, pyuria, flank pain, and decreased urine output infarction in brain: hemiparesis, aphasia, and other neurologic deficits infarction in lung: cough, pleuritic pain, pleural friction rub, dyspnea and hemoptysis peripheral vascular occlusion: numbness and tingling in an arm, leg, finger, or toe, or signs of impending peripheral gangrene
SIGNS OF VENOUS INSUFFICIENCY IN THE LOWER EXTREMITIES
Skin color reddish brown or cyanotic if extremity lowered Normal temperature Normal pulse Often marked edema, usually foot to calf Brown pigmentation around ankles Diagnostics health history lab data CBC - elevated WBC blood cultures - positive for microbe ESR - elevated CXR - to detect heart failure or cardiomegaly transesophageal echocardiogram to detect vegetation and abscesses on valves EKG to detect dysrhythmias Management - Clients at risk for prosthetic valves prophylaxis - to prevent endocarditis; i.e. MVP, cardiac lesions antibiotics - to treat underlying infection antipyretics - to control fever anticoagulants - to prevent embolization oxygen - to prevent tissue hypoxia surgical - possible valve replacement
Endocarditis Nursing interventions the Cardio-Care Six THE CARDIO-CARE SIX: A,B,C,D,E,F ADL: Help the client with activities of daily living and how to schedule activities that minimize cardiac stress. Bed rest Commode at bedside (it is less stressful to the heart than using a bedpan) Diversions: offer diversions that don't stress the heart (e.g., no hand-held electronic games). Elevate head of bed or sit client up to a position of comfort. Feelings: plan time for the client to express his
Endocarditis
watch for signs of infiltration or inflammation at venipuncture site; rotate sites according to agency policy
reinforce client and family teaching regarding: explanation of all procedures in a simple and culturally sensitive manner involvement of the client and family in scheduling the daily routine activities and allowing client and family to participate in care relaxation techniques (meditation, visualization, or guided imagery) to cope with stress, pain, or insomnia endocarditis and the need for long-term therapy the need for prophylactic antibiotics before dental work and other invasive procedures to report fever, tachycardia, dyspnea and sudden shortness of breath.
Rheumatic heart disease (rheumatic endocarditis)
Definition and related terms
rheumatic heart disease - damage to the heart by one or more episodes of rheumatic fever. Pathogen is a group A streptococci.
rheumatic endocarditis - damage to the heart, particularly the valves, resulting in valve leakage (regurgitation) and/or stenosis. To compensate, the heart's chambers enlarge and walls thicken.
Rheumatic heart disease (rheumatic endocarditis)
Epidemiology
worldwide, millions of new cases of rheumatic fever are reported each year rheumatic fever follows a group A streptococcal infection - prevention is simply to find and treat streptococcal pharyngitis with malnutrition and crowded living, rheumatic fever is most common in children between the ages of five and 15 rheumatic fever strikes most often during cool, damp weather. In the U.S., it is most common in the northern states no one knows how and why group A streptococcal infections cause the lesions called Aschoff bodies damage depends on site of infection: most often the mitral valve in females and the aortic valve in males malfunction of these valves leads to severe pericarditis, and sometimes pericardial effusion and fatal heart failure. Of those who survive this complication, about 20% die within ten years
Rheumatic heart disease (rheumatic endocarditis)
Findings
streptococcal pharyngitis
sudden sore throat
throat reddened with exudate
swollen, tender lymph nodes at angle of jaw
headache
fever to 104 degrees Fahrenheit
polyarthritis manifested by warm and swollen joints
carditis
chorea
erythema marginatum (wavy, thin red-line rash on trunk and extremities)
subcutaneous nodules
fever to 104 degrees Fahrenheit
heart murmurs pericardial friction rub and pericardial rub
no lab test confirms rheumatic fever, but some support the diagnosis
Rheumatic heart disease (rheumatic endocarditis)
Diagnostics antistreptolysin 0 titer (ASO titer) - increased ESR - increased throat culture - positive for streptococci WBC count - increased RBC parameters - normocytic, normochromic anemia C-reactive protein - positive for streptococci
Management give antibiotics on schedule to maintain blood levels provide analgesics - for pain/inflammation PRN oxygen to prevent tissue hypoxia surgical - commissurotomy, valvuloplasty, prosthetic heart valve
Rheumatic heart disease (rheumatic endocarditis)
Nursing Interventions the cardio-care six assist the client with chorea in grasping objects; prevent falls encourage family and friends to spend time with client and fight boredom during the long, tedious convalescence reinforce client and family teaching regarding: explanation of all tests and treatments nutritional needs hygienic practices resumption of ADLs slowly and scheduling rest periods to report penicillin reactions: rash, fever, chills
to report findings of streptococcal infection sudden sore throat diffuse throat redness and oropharyngeal exudate swollen and tender cervical lymph glands pain on swallowing temperature of 101 to 104 degree Fahrenheit headache nausea the avoidance of crowds and people with respiratory infections explanation of the necessity of long-term antibiotics actions to cope with the temporary chorea
Valve Disorders
Mitral stenosis Definition - Mitral valve thickens, with result of narrowing passageway and blocking blood flow from the left atrium to the left ventricle during atrial systole Epidemiology of clients with mitral stenosis, two-thirds are female most cases of mitral stenosis are caused by rheumatic fever Findings mild - no symptoms moderate to severe dyspnea on exertion paroxysmal nocturnal dyspnea (PND) orthopnea weakness, fatigue,
complications peripheral and facial cyanosis in severe cases jugular vein distention with severe pulmonary hypertension or tricuspid stenosis - ascites edema hepatomegaly diastolic thrill at the cardiac apex
Valve Disorders
Diagnostics
history and physical exam
EKG - for changes of left atrial enlargement and right ventricle enlargement
echocardiogram - for restricted movement of the mitral valves and diastolic turbulance
Management
antidysrhythmics as indicated
if medication fails, atrial fibrillation is treated with cardioversion.
low-sodium diet - to control underlying heart disease
oxygen if needed - to prevent hypoxia
surgery - mitral commissurotomy or valvotomy
Valve Disorders
Nursing interventions the Cardio-Care Six watch closely for findings of heart failure, pulmonary edema and reactions to drug therapy if client has had surgery, watch for hypotension, dysrhythmias, and thrombus formation monitor The Cardio Seven reinforce client and family teaching regarding: TEACH THE CARDIO FIVE: TDDDS Tests and treatments: discuss them in simple, culturally sensitive ways. Drugs, their side effects, how long client will take them, and their expected effects. Diet: balanced nutrition and restrictions (such as low sodium). Disease, its management, when and what signs to report promptly: the 'watchfor s '. Smoker? Stress benefits of stopping smoking, minimization of other stimulants - caffeine, chocolate, nonprescription drugs, herb cautions explaination of the need for long-term antibiotic therapy and the need for additional antibiotics before dental care or any invasive procedure the need to report early findings of heart failure such as dyspnea or a hacking, nonproductive cough