RESPIRATORY SYSTEM Billy Ray A. Marcelo, RN Faculty Bataan Peninsula State University
OVERVIEW
Basic Process of Oxygenation Ventilation- degree of compliance, airway resistance, accessory muscles (Respiratory System) Diffusion- thickness of membranes (Hematologic System) Perfusion- integrity of transport system (CV and Hematologic System)
OVERVIEW
Function: Respiratory System Obtains O2, removes CO2 Filters particles from incoming air Control T and water content Role in sense of smell Regulates blood pH
OVERVIEW
OVERVIEW
Upper Respiratory Tract Filtering of air, warming, moistening Humidification A. Nose Framework of cartilage 2 septum/nostril Anastomosis of capillaries (Keisselbach) B. Pharynx (Throat)- organ of GI and RT Muscular pasageway for food and air Nasopharynx Oropharynx Laryngopharynx
OVERVIEW C. Larynx (Voicebox) Phonation (speech production) Cough reflex Frameworks Arythenoid and cricoid cartilage Thyroid gland Hyoid bone Glottis Epiglottis Opens: passage of air Closes: passage of food
Upper Respiratory Tract
OVERVIEW
Lower Respiratory System Gas exchange A. Trachea (windpipe) Cartilaginous rings, ‘U’ shape Site of permanent artificial airway (tracheostomy) B. Carina- area of bifurcation of brochi C. Bronchi R main bronchus- wider straighter L main bronchus
OVERVIEW
Lower Respiratory System D. Lungs- covered by serous membrane R- 3 lobes L- 2 lobes Pleural cavity Parietal- with 20 cc of fluid to prevent friction, with nerve endings Visceral- without nerve endings Pleural
OVERVIEW
Lower Respiratory System D. Lungs Terminal bronchioles Alveoli (Acinar cells)- site of gas exchange (CO2 and O2) Type II cells- secretes SURFACTANT (phospholipid lipoprotein) → ↓ surface tension
PNEUMONIA
Inflammation of lung parenchyma → pulmonary consolidation as the alveoli are filled with exudates Causative Agents Streptococcus pneumoniae (Pneumococcal pneumonia) Hemophilus influenzae (Bronchopneumonia) E. Coli Klebsiella Pseudomonas aeruginosa
PNEUMONIA
Predisposing Factors:
PNEUMONIA Excessive smoking Air pollution Over fatigue Prolonged immobility → hypostatic pneumonia Aspiration Immunocompromised state AIDS- Pneumocystis carinii (taking Zidovudine or AZT) Bronchogenic CA
Signs and Symptoms: PNEUMONIA
Productive cough (greenish to rusty sputum) Dyspnea with prolonged expiratory grunt Fever, chills, anorexia, N/V, weight loss Pleuritic friction rub Rales, crackles, Bronchial wheezing Cyanosis Chest pain Abdominal distension → paralytic ileus (most feared Cx)
Diagnostic Procedures: PNEUMONIA
Sputum C/S Gram-staining C/S Chest X-Ray- reveals pulmonary consolidation ABG- ↓ pO2 CBC- ↑ WBC, ↑ ESR
Nursing Management: PNEUMONIA
CBR Place pt. on semi-fowler’s Low flow O2 as ordered
Give comfortable and humid environment Diet: ↑ CHON, ↑ CHO, ↑ Vit C
Force fluids to liquefy secretions Importance of receiving immunization as recommended
Nursing Management: PNEUMONIA
Administer meds as ordered Broad spectrum antibiotics Penicillin, Tetracycline, Macrolides Antipyretics Mucolytics/ Expectorants (Guiafenesin, Glycerine, Guiacolate)
Nursing Management: PNEUMONIA Institute pulmonary toilet DBE Coughing Chest physiotherapy (CPT) Turning and repositioning Nebulize and suction prn
Chest Physiotherapy
Nursing Management: PNEUMONIA
Institute postural drainage as ordered Pt is placed on various positions to promote drainage of secretions, stay for 20-30 minutes Best done before breakfast, or 2-3 hrs p.c. Pt. should be well hydrated, knows how to cough Prone with pillow on abdomen- drains lower part of the lungs Supine with buttocks up- drains upper part of the lungs
Postural Drainage
Postural Drainage
Nursing Management: PNEUMONIA
Institute postural drainage as ordered Monitor VS, breath sounds Administer bronchodilators 15-30 minutes prior Encourage DBE Stop if pt can’t tolerate the procedure Give oral care post procedure No to pt with: hemoptysis, unstable VS, ↑ ICP, ↑ IOP
Nursing Management: PNEUMONIA Discharge Health Teaching Stop smoking Regular adherence to meds Dietary modification Follow-up care Prevent Cx: atelectasis and meningitis
PULMONARY TUBERCULOSIS
Or Koch’s Disease Causative agent: MTB- acid-fast, non-motile Predisposing Factors Malnutrition Overcrowding Alcoholism Ingestion of affected cattle (with M. bovis) Virulence of the microorganism
PULMONARY TUBERCULOSIS
Signs and Symptoms: PTB Productive cough- yellowish secretions > 2 wks Dyspnea Low grade afternoon fever- Pathognomonic Sign Night sweats- Classical Sign Anorexia, general body malaise Weight loss Chest pain Hemoptysis
Diagnostic Procedures: PTB
Mantoux Test- skin test, injection of PPD Reading: after 48-72 hrs (+) exposure to PTB: DOH: 8-10 mm induration WHO: 10-14 mm induration
Diagnostic Procedures: PTB Sputum AFB(+) MTB Chest X-raypulmonary infiltrates (caseous necrosis) CBC- ↑ WBC
PULMONARY TUBERCULOSIS
Nursing Management CBR Comfortable environment O2 inhalation as ordered Force fluids to liquefy secretions NO CPT, only DBE and coughing Nebulize and suction prn Place on semi-fowler’s Diet: ↑ CHON, ↑ CHO, ↑ Vit C
PULMONARY TUBERCULOSIS
Short Course Chemotherapy I. Intensive Phase INH- given for 4 mos., taken a.c. S/E: peripheral neuritis- give Vit B6 Rifampicin- given for 4 mos., taken a.c. S/E: all body secretions turned red-orange PZA- given for 2 mos., taken p.c. S/E: skin rashes, nephro and hepatotoxicity PZA is replaced by Ethambutol S/E: optic neuritis (visual disturbance)
PULMONARY TUBERCULOSIS
Short Course Chemotherapy II. Standard Regimen Streptomycin IM (Aminoglycoside) S/E: Ototoxicity due to damage to CN VIII→ temporary hearing loss Nephrotoxicity- monitor BUN and Crea levels
PULMONARY TUBERCULOSIS
Discharge Health Teaching Avoid precipitating factors Take meds religiously If missed 1 day’s meds, NEVER ↑ the dose on the next day, simply let the pt continue taking the meds Prevent Cx: Atelectasis and Miliary TB Follow-up care
HISTOPLASMOSIS
Acute fungal infection characterized by inhalation of contaminated dust with Histoplasma capsulatum from bird’s manure
HISTOPLASMOSIS
S/Sx: PTB, Pneumonia-like Productive cough Dyspnea Cyanosis Hemoptysis Fever, chills, anorexia, general body malaise Chest and joint pain
HISTOPLASMOSIS Diagnostic Procedure (+) Histoplasmin skin test (+) agglutination test ↑ WBC ABG- ↓ pO2 CXR- (+) infiltrates
HISTOPLASMOSIS
Nursing Management CBR, semi Fowler’s position O2 inhalation as ordered Force fluids Encourage coughing & DBE Nebulize and suction prn
HISTOPLASMOSIS
Nursing Management Administer meds as ordered Antifungal agent: Amphotericin B (Fungizone) S/E: nephrotoxicity and hypoK+ Corticosteroids Antipyretics Antihistamines Mucolytics/expectorants
HISTOPLASMOSIS
Nursing Management Spraying of breeding places Prevent Cx: Atelectasis and Bronchietasis
SEVERE ACUTE RESPIRATORY SYNDROME (SARS)
Cause: Coronavirus Begins with fever, body aches, mild respiratory Sxs After 2-7 days, dry cough & dyspnea develops MOT: close person-to-person contact (direct contact with infectious secretions and soiled articles) Prevention: avoiding contact with those suspected of having SARS, avoiding travel to countries with SARS outbreak, frequent hand washing
INHALATION INJURIES: CO poisoning
CO: colorless, Blood odorless, tasteless, Level with affinity for Hgb 1-10% 200X greater than 11-20% O2, forming carboxyHgb→ tissue 21-30% hypoxia
Assessment Impaired visual acuity Flushing, HA N/, impaired dexterity
31-40% Vom,dizziness,syncope 41-50% ↑HR, ↑RR >50%
Coma, death
INHALATION INJURIES: CO poisoning
Interventions Remove victim from exposure Administer 100% O2 Assess need for CPR Monitor VS and CO levels
OCCUPATIONAL LUNG DISEASE: SILICOSIS Or Asbestosis or Coal Workers’ Pneumoconiosis Fibrotic lung disease caused by inhalation or organic dusts over long periods of time Common among miners & sandblasters
OCCUPATIONAL LUNG DISEASE: SILICOSIS
S/Sx Uncomplicated or simple: asymptomatic with evidence of fibrosis on CXR Chronic complicated: malaise, A/, wt loss, severe dyspnea on exertion, massive fibrosis on CXR
OCCUPATIONAL LUNG DISEASE: SILICOSIS Interventions Eliminate the toxic substance O2 as ordered Coughing and DBE Administer antitussives for cough & anti-TB meds as ordered (Cx: PTB)
COPD
Types Chronic Bronchitis Bronchial Asthma Bronchiectasis Pulmonary Emphysema
COPD-Chronic Bronchitis
Inflammation of bronchi→ hyperplasia of goblet mucusproducing cells → narrowing of smaller airways
COPD-Chronic Bronchitis
Predisposing Factors Excessive, chronic smoking Air pollution
COPD-Chronic Bronchitis
S/Sx Productive cough Dyspnea at exertion Prolonged expiratory grunt Scattered rales, rhonchi Anorexia, general body malaise Cyanosis- Blue Bloaters Feeling of breathlessness Pulmonary HTN leading to peripheral edema and Cor Pulmonale (most feared Cx)
Cor Pulmonale
COPD-Bronchial Asthma Reversible inflammatory disorder of lung tissue due to hypersensitivity to allergens → narrowing of smaller airways Predisposing Factors (based on 3 types) Extrinsic (Atopic/Allergic) Pollen, dust, furs, fumes, gases, smoke, danders, lints
COPD-Bronchial Asthma
Intrinsic (Non-Atopic/Non-Allergic) Hereditary Drugs: ASA, Pen, Phenylbutazone, Beta blockers Foods: seafoods, eggs, chicken, chocolate, milk and its products Food additives- nitrates (also can cause CA) Sudden change in T, air pressure and humidity Extreme emotion Physical stress Mixed- combination of the 2 Most common type (90% of cases)
Pathophysiology Allergens Release of IgE by B-lymphocytes IgE + mast cells (respiratory tract) Damage to mast cells Release of chemical mediators (histamine, bradykinin, serotonin, prostaglandin)
vasodilatation Hypotension blood congestion shock increased capillary permeability escape of colloids edema decreased blood vol.
COPD-Bronchial Asthma
COPD-Bronchial Asthma
S/Sx Non-productive cough Dyspnea Wheezing on expiration Slight cyanosis Mild restlessness and apprehension Tachycardia and palpitation Diaphoresis
COPD-Bronchial Asthma
Diagnostic Procedures ABG- ↓pO2 PFT- ↓ vital lung capacity (max. vol. of air that can be exhaled with the deepest breath possible)
COPD-Bronchial Asthma
Nursing Management Administer meds as ordered Bronchodilators- inhalation or metered dose inhaler (pump) Corticosteroids Mucolytics/Expectorants Anti-histamine O2 as ordered
COPD-Bronchial Asthma
COPD-Bronchial Asthma
Nursing Management Force fluids Nebulize and suction prn Comfortable and humid environment
COPD-Bronchial Asthma
Discharge Health Teaching Avoid precipitating factors Regular adherence to meds Sudden withdrawal to corticosteroids→ status asthmaticus Prevent Complications Emphysema Status asthmaticus- Drug of Choice: Epinephrine
COPD-Bronchiectasis
Permanent dilatation of bronchus → destruction of elastic and muscular tissues of the alveolar walls
COPD-Bronchiectasis
Predisposing Factors Recurrent URTI and LRTI Congenital anomalies Lung tumor Signs and Symptoms Productive cough Dyspnea Anorexia, general body malaise Cyanosis Hemoptysis
COPD-Bronchiectasis Diagnostic Procedures ABG- ↓ pO2 Bronchoscopy
COPD-Bronchiectasis
Bronchoscopy: Nursing Management Pre-op: informed consent, maintain on NPO, monitor VS Post-op Feed when gag reflex returns Avoid talking, coughing, smoking → chronic irritation Monitor for S/ of gross/frank bleeding WOF laryngospasm and edema → DOB, SOB → prepare trache set at bedside
COPD-Bronchiectasis
Nursing Management Same as in pulmonary emphysema Assist in surgical procedure Pneumonectomy Position post-op: lie on affected side Segmental wedge lobectomy Position post-op: lie on unaffected side
Pneumonectomy vs. Lobectomy
COPD-Pulmonary Emphysema
Irreversible, end-stage stage of COPD characterized by inelasticity of alveolar wall → air trapping → maldistribution of gases → over distension of thoracic cavity → ↑ A:P diameter (Barrel-chest)
COPD-Pulmonary Emphysema
COPD-Pulmonary Emphysema
Predisposing Factors Excessive, chronic smoking Allergy Air pollution Hereditary- deficiency of alpha-1 antitrypsin → elastase/elastin → alveolar recoil (Northern European origin) Elderly- high risk group
COPD-Pulmonary Emphysema
Types Centrilobular/Panlobular Blue Bloaters pCO2 ↑, pO2 ↓, resp. acidosis with hypoxemia Centriacinar/Panacinar Pink Puffers pCO2 ↓, pO2 ↑, resp. alkalosis with hyperoxemia
Blue Bloater vs. Pink Puffer
The Blue Bloater
COPD-Pulmonary Emphysema
Signs and Symptoms Productive cough Dyspnea at rest Anorexia, general body malaise On lung percussion- resonance to hyperresonance
COPD-Pulmonary Emphysema
Signs and Symptoms (+) nasal flaring rales, rhonchi ↓ breath sounds, vocal fremiti Barrel chest- Pathognomonic Sign (+) pursed-lip breathing
COPD-Pulmonary Emphysema
Nursing Management CBR Administer meds as ordered Bronchodilator Corticosteroid Antibiotics Mucolytics/expectorants
COPD-Pulmonary Emphysema
Nursing Management Low flow, Fixed concentration O2 inhalation as ordered not to remove the Hypoxic Drive
COPD-Pulmonary Emphysema
Nursing Management Force fluids Diet: ↑ CHON, ↑ Vit & min., ↓ CHO DBE- pursed-lip, cascade coughing, CPT Nebulize and suction secretions prn
COPD-Pulmonary Emphysema
Discharge Health Teaching Stop smoking Regular adherence to meds Prevent Complications Atelectasis Cor Pulmonale CO2 narcosis- severe disorientation/confusion → coma Pneumothorax Follow-up care
Restrictive Lung Disorders Pneumothorax Partial/complete collapse of the lungs due to accumulation of air in the pleural space
Pneumothorax
3 types Spontaneous- without obvious cause e.g. rupture of bleb (alveolar fluid sac) in recurrent lung inflammation and infection Open- thru chest opening e.g. stab, gunshot wounds Tension- from blunt chest injury or from mech. vent. With PEEP air enters pleural space with each inspiration and cannot escape →↑ thoracic cavity → mediastinal shift E.g. flail chest (with paradoxical breathing)
Pneumothorax
Predisposing Factors Chest trauma Recurrent inflammatory lung condition Lung tumors
Pneumothorax
Signs and Symptoms Cool, moist skin (beginning of shock) Sharp, chest pain Unexplained dyspnea ↓ breath sounds → lung collapse ↓ lung expansion Cyanosis
Pneumothorax
Signs and Symptoms Mild restlessness/apprehension On lung percussion- resonance to hyperresonance SQ emphysema (crepitus on palpation) Tracheal deviation to unaffected side
Pneumothorax
Diagnostic Procedure ABG- ↓ pO2 Chest X-ray- partial or complete lung collapse
Nursing Management: Pneumothorax Assist in intubation Administer meds as ordered Narcotic analgesic Antibiotics
Nursing Management: Pneumothorax
Assist in thoracentesis/ chest tube thoracostomy Remove air- insert at 2nd-3rd ICS Remove fluid- insert laterally near base, posteriorly at 8th-9th ICS Pt position: struggling to a chair, pt exhales and hold breath during insertion (under local anesthesia)
Thoracentesis
Nursing Management: Pneumothorax Attach tube to water-seal drainage Objectives: To reestablish (-) pressure in the lungs To promote lung expansion To drain air, fluid and blood and to prevent it reflux
Nursing Management: Water Seal Drainage Monitor VS, I/O, breath sounds DBE Administer meds as ordered Maintain strict asepsis
Nursing Management: Water Seal Drainage Prepare at bedside: vaselinized gauze Hemostan clamp Extra bottle with water
Nursing Management: Water Seal Drainage
Monitor for oscillation and fluctuation N- (+) intermittent bubbling, ↑ with inspiration, ↓ expiration Check for leakage If (-) bubbling: check for kinks, obstructionmilk towards drainage bottle, or lungs are fully expanded
Water Seal Drainage
Nursing Management: Water Seal Drainage 3 parameters to remove chest tube (-) bubbling/fluctuations (+) symmetrical breath sounds Chest X-ray confirms full lung expansion
Nursing Management: Water Seal Drainage
Before, During and After Removal of Chest Tube Encourage DBE Monitor VS, breath sounds Give analgesic prior to removal Instruct pt to perform Valsalva maneuver for easy removal and to prevent air entry to pleural space Apply vaselinized occlusive dressing, WOF bleeding
PLEURAL EFFUSION
Collection of fluid in the pleural space S/Sx
Pleuritic pain that is sharp & ↑ with inspiration Dyspnea on exertion Dry, nonproductive cough caused by bronchial irritation or mediastinal shift ↑HR, ↑T ↓ breath sounds CXR: confirms the dx & shows mediastinal shift
PLEURAL EFFUSION
Interventions Identify & tx the underlying cause Monitor breath sounds High Fowler’s position Coughing & DBE Prepare the pt for thoracentesis
PLEURAL EFFUSION Interventions If recurrent, prepare the pt for: Pleurectomy: surgically stripping parietal away from visceral pleura to promote adhesion of the 2 layers during healing Pleurodesis: instilling sclerosing substance into pleural space via thoracotomy tube
EMPYEMA Collection of pus in the pleural cavity (thick, opaque, foul-smelling) Causes: pulmonary infection, lung abscess due to thoracic surgery or chest trauma Goal of tx: emptying empyema cavity, reexpanding the lung, controlling infection
EMPYEMA S/Sx of infection + ↓ chest wall mov’t & pleural exudate on CXR Interventions
Semi or High Fowler’s position Monitor breath sounds Coughing and DBE Splint the chest if in pain Antibiotics as ordered
EMPYEMA
Interventions Assist in chest tube insertion If (+) marked pleural thickening, prepare the pt for Decortication: surgical removal of restrictive mass of fibrin & inflammatory cells
PLEURISY
Inflammation of the visceral & parietal pleura, rubbing together during breathing causing pain May be caused by pulmonary infarction or pneumonia Usually occurs on one side of the chest (lower lateral portion)
PLEURISY
S/Sx Knifelike pain aggravated by deep breathing & coughing Dyspnea Pleural friction rub on auscultation Apprehension
PLEURISY
Interventions Identify and tx the cause Monitor breath sounds Hot or cold applications as ordered Encourage coughing & DBE Lie on affected side to splint the chest Analgesics as ordered
ACUTE RESPIRATORY DISTRESS SYNDROME
A form of acute respiratory failure as a complication of other condition, caused by diffuse lung injury→ extravascular lung fluid →compression of terminal airways → ↓ lung vol. & compliance ABG= resp. acidosis & hypoxemia not responding to ↑ O2 concentration CXR= interstitial edema
ACUTE RESPIRATORY DISTRESS SYNDROME
Predisposing factors Sepsis Fluid overload Shock Trauma Neuro injuries Burns DIC Drug ingestion Toxic substance inhalation
ACUTE RESPIRATORY DISTRESS SYNDROME
S/Sx ↑ HR Dyspnea ↓ breath sounds Deteriorating blood gas levels Hypoxemia despite high O2 concentration ↓ pulm. compliance Pulm. infiltrates
ACUTE RESPIRATORY DISTRESS SYNDROME
Interventions Identify & tx the cause O2 as ordered High Fowler’s position Fluid restriction as ordered Diurretics, anticoagulants, corticosteroids as ordered Prepare for intubation and mechanical ventilation with PEEP
MECHANICAL VENTILATION
TYPES 1. Pressure-cycled The ventilator pushes air into the lungs until an airway pressure is reached Used for short periods (pt in PACU & for respiratory tx)
2. Time-cycled With preset time Used for pedia & neonatal pt
MECHANICAL VENTILATION
3. Volume-cycled With preset tidal volume that is delivered regardless of the changing lung compliance or airway resistance (from the vent. or from the pt) 4. Microprocessor Built into the vent. to allow continuous monitoring of the vent. functions, alarms & other parameters For pt with severe lung disease or required prolonged weaning
MECHANICAL VENTILATION MODES 1. Controlled With set TV & RR For pt who cannot initiate respiratory effort Least used bec. if the pt attempts to breathe, the vent. blocks the effort
MECHANICAL VENTILATION
MODES 2. Assist-control
With set TV while allowing the pt control the RR Most commonly used The vent. takes over the work of breathing for the pt Responds to pt’s inspiratory effort If the pt’s spontaneous RR ↑ the vent. continues to deliver preset TV→ hyperventilation & respiratory alkalosis
MECHANICAL VENTILATION
MODES 3. Synchronized Intermittent Mandatory Ventilation (SIMV)
With set TV and RR while allowing the pt control own TV & RR in between the vent. breaths Used as a primary vent. mode or as a weaning mode The no. of SIMV breaths is ↓ gradually until the pt gradually resumes spont. breathing
MECHANICAL VENTILATION
CONTROLS & SETTINGS 1. TV: vol. of air that the pt receives with each breath 2. RR: no. of vent. breaths/min 3. Fraction of inspired O2 (FiO2): O2 concentration delivered to the pt; determined by pt’s condition & ABG 4. Sighs: vols. of air 1.5-2X the set TV, delivered 6-10X/hr, prevents atelectasis
MECHANICAL VENTILATION CONTROLS & SETTINGS 5. Peak airway inspiratory pressure (PIP) Pressure needed by the vent. to deliver set TV at a given compliance Reflects changes in compliance of the lungs & resistance in the vent. or the pt
MECHANICAL VENTILATION
CONTROLS & SETTINGS 6. Continuous positive airway pressure (CPAP) Applied throughout the entire respiratory cycle for spont. breathing pt Keeps the alveoli open during inspiration & prevents alveolar collapse Used primarily as a weaning modality since no vent. breaths are delivered, only FiO2 RR is determined by the pt’s efforts
MECHANICAL VENTILATION
CONTROLS & SETTINGS 7. Positive end-expiratory pressure (PEEP) Exerted during the expiratory phase of ventilation Improves oxygenation by enhancing gas exchange & preventing atelectasis Used in pt with severe gas exchange disturbance Higher amounts of PEEP (↑15) ↑ the chance of Cx: barotrauma tension pneumothorax
MECHANICAL VENTILATION
CONTROLS & SETTINGS 8. Pressure Support Application of positive pressure on inspiration Eases workload of breathing May be used in combo with PEEP as a weaning method
Nursing Interventions: MECHANICAL VENTILATION Assess the pt first before the ventilator Assess for VS, breath sounds, respiratory status & breathing patterns Monitor skin color, pulse oximetry, ABG Suction secretions prn Assess the ventilator settings
Nursing Interventions: MECHANICAL VENTILATION Check level of water in humidifier & temp. or humidification system Ensure that alarms are set If cause of alarm cannot be determine, manually ventilate the pt Empty ventilator tubing when moisture collects
Nursing Interventions: MECHANICAL VENTILATION
Weaning: the process of going from ventilator dependence to spont. Breathing SIMV mode T-piece Decreasing PS
Nursing Interventions: MECHANICAL VENTILATION Causes of Alarms High-Pressure Alarm
↑ secretions Wheezing, bronchospasm Displaced, kinked, ET Excess water in vent. Tubings Pt. coughs, gags, bites ET Pt is anxious, fighting the vent.
Nursing Interventions: MECHANICAL VENTILATION Causes of Alarms Low-Pressure Alarm
Disconnection or leak in the ventilator Busted airway cuff Pt stops spontaneous breathing
Nursing Interventions: MECHANICAL VENTILATION
WOF Cx
Hupotension r/t (+) pressure increasing intrathoracic pressure Pneumothorax SQ emphysema Stress ulcers Malnutrition Infection Muscular atrophy Ventilator dependence, inability to wean
CGFNS/NCLEX Question
A pt in the ER department has multiple fractured ribs and R-sided tension pneumothorax. The RN should expect to prepare the pt for which of the following procedures? A. Electrocardiography B. Urinary catheter placement C. Chest tube insertion D. Gastric lavage
CGFNS/NCLEX Question
A pt with asthma is producing thick, white secretions. Which of the following nursing measures would be most appropriate for the RN to include in her plan of care? A. increase fluid intake B. promote exercise C. administer O2 D. encourage coughing
CGFNS/NCLEX Question
A RN education a pt on the correct use of metered dose inhaler should instruct the pt to A. take several short shallow breaths before inhaling B. hold breath after inhaling the drug C. cough before inhaling the drug D. press the cartridge down before inhaling the drug
CGFNS/NCLEX Question
The MD orders a corticosteroid inhaler 4 puffs BID for a pt with asthma. Which of the following actions by the pt should indicate to the RN that the pt needs further teaching? A. taking 4 puffs in rapid succession B. pausing for 1-2 mins. in between puffs C. rinsing the mouth with water after inhaling D. inhaling meds slowly
CGFNS/NCLEX Question
A pt who has asthma is given instructions about the use of inhalant meds. Which of the following statements, if made by the pt, indicates that the pt understands the instructions?
CGFNS/NCLEX Question A. “I will use the steroid inhaler 1 hr before I use the brochodilator.” B. “I will use the bronchodilator before I use the steroid inhaler.” C. “I need to take these meds 1 hr after each meal.” D. “I need to alternate the sequence of inhaler administration.”
CGFNS/NCLEX Question
An asthmatic pt has orders for all of the following meds. Which meds should a RN expect to prepare when the pt shows signs of status asthmaticus? A. Epinephrine (Adrenaline) B. Theophylline (Theo-Dur) C. Erythromycin (Robimycin) D. Cromolyn Sodium (Nasalcrom)
CGFNS/NCLEX Question
Cromolyn Sodium (Intal) is ordered for a pt who has asthma. A RN would determine that the pt understands when to take the med if the pt makes which of the following statements? A. “I will use my inhaler after meals.” B. “I will use my inhaler prior to exercise.” C. “I will use my inhaler when I am having an attack.” D. “I will use my inhaler after being outside in cold weather.”
CGFNS/NCLEX Question
Prior to discharging a pt who is asthmatic, a RN should include which of the following measures in the teaching plan? A. Discussing techniques for weight control while taking steroids B. Identifying specific environmental triggers C. Maintaining school performance using a home tutor D. Keeping a record of weekly sputum testing
CGFNS/NCLEX Question
A RN teaches pursed-lip breathing to a pt who has COPD. Which of the following statements indicates the pt understands the instructions? A. “I will maintain a supine position during the exercise.” B. “I will alternate positions during the exercises.” C. “I will exhale for twice as long as I inhale.” D. “I will inhale and exhale thru my nose.”