Pathophysiology-maia

  • April 2020
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PATHOPHYSIOLOGY (Schematic Diagram) Precipitating Factor:

Predisposing Factor:

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HYPERTENSION

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AGE

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STRESS

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GENDER

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DIABETES MELLITUS

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FAMILY HISTORY

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HISTORY OF STROKE

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HYPERLIPIDEMIA

Increased blood flow to the brain

Compromise of the integrity of cerebral arterioles Deposition of lipid and hyaline material in the arterial wall Weakening of the walls Formation of Chalcot- Bouchard aneurysm -FEVER

Rupture of the anterior cerebral artery

LEUKOCYTOS

Inflammation of the frontal lobe -ABG, CEREBRAL Alteration in the ANGIOGRAPHY, MRI, CT SCAN, Subarachoid screw, cerebral ventriculostomy, epidural ICP component monitoring. -RESTLESSNESS, CHANGES IN LOC, DROWSINESS,CONFUSION,

Bleeding or hemorrhage on at the affected area Hematom a formation

Presence of free blood in the interstitial areas

Thrombu s formatio

Increase in the Intracranial pressure Metaboli c regulatio

Compressi on of the brain componen

Auto regulatio n -PROJECTI LE VOMITING, NUMBNES S OF EXTREMIIT IES , VISUAL DISTURBA

Decreas e oxygen supply Stimulate s further swelling and inflammat Ischemi a Scar Formatio n X RAY, CT scan, MRI

-BROCA’S APHASIA, HEMIPARESIS, HEMIANOPSIA , DYSARTHRIA, INCAPABLE LEGEND: OF ABSTRACT THINKING

RED FONT SYMPTOMS PINK FONT

Compensato ry mechanism

IF NOT TREATED

Cellular edema

Stimulation of vasomotor centers

Alteration in the frontal lobe function

Increas e system ic

Cell membrane destruction

CT SCAN AND MRI

Vasospas m

Electroly te imbalan

Acidosi s

-SERUM ELECTROLYTE TEST (decrease Mg, K), PE/PA, ABG(Respiratory alkalosis) -CHANGES IN THE V/S

-VITAL SIGN CHANGES

IF TREATED

Management: Brain stem herniation

SURGICAL: craniectomy, craniotomy, AVMS

stupor MEDICAL: Pharmacologic Therapy - Comatose SIGNS AND SYMPTOMS AMD THE Fair (citicholine, mannitol, dilatin, DIAGNOSTIC EXAMS Prognosis captopril, Nitroglycerin, furosemide, - DISEASE PROCESS remopain, Kalium Durule, MgSO4) Worsening - ofPOSSIBLE INTERVENTIONS AND the condition -

DEATH ACTUAL INTERVENTIONS AND