Medical Surgical Nursing A
Care of the Patient With a Neurological Disorder
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Anatomy and Physiology Central nervous system (CNS)
Peripheral nervous system
Brain Spinal cord
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Somatic (voluntary) Autonomic (involuntary)
Anatomy and Physiology Neurons
Glial cells
Transmitter cells Carry messages to and from brain and spinal cord
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Support and protect neurons Produce cerebral spinal fluid
Anatomy and Physiology CNS: brain
Cerebrum – lobe functions Diencephalon – thalamus, hypothalamus Cerebellum – balance, coordination Brain stem – midbrain, pons, medulla oblongata
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Anatomy and Physiology PNS: Somatic (voluntary)
31 pairs of spinal nerves 12 pair of cranial nerves
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Anatomy and Physiology PNS: Autonomic (involuntary)
Controls: Smooth
Muscles Cardiac Muscles Glands
Check and balance system: Sympathetic
nervous system Parasympathetic nervous system BGallatin 2007
Neurological Assessment History
Headaches Loss of function Visual acuity Seizures Numbness
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Pain Personality change Mood swing Fatigue
Neuro Assessment Mental Status
Orientation Mood and behavior General knowledge Short term memory Long term memory
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Neuro Assessment Level of consciousness Glasgow Coma Scale
Eye opening Verbal response Motor response
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Neuro Assessment Language and Speech
Aphasia Sensory Expressive Global
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Cranial Nerves
I. Olfactory II. Optic III. Oculomotor IV. Trochlear V. Trigeminal VI. Abducens
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VII. Facial VIII. Acoustic IX. Glossopharyngeal X. Vagus XI. Spinal Accessory XII. Hypoglossal
Neuro Assessment Motor Function
Paralysis Paresis Flaccid Spastic
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Neuro Assessment Sensory and Perceptual Status
Pain Touch Temperature Proprioception Unilateral neglect Hemianopia BGallatin 2007
Neuro Assessment Blood and urine ABG Lumbar puncture Imaging EEG EMG Carotid Duplex BGallatin 2007
Neurological Problems Headache
Vascular – migraine, cluster, hypertensive Tension – stress Traction-inflammatory – infection, occlusion vessels
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Neurological Problems Increased Intracranial Pressure (IIP) Occurs slowly or rapidly May lead to brain stem herniation and death
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Assessment of IIP Subjective
Diplopia Personality change Thought processes change Headache Nausea
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Assessment of IIP Objective Decreasing LOC
Hyperthermia Weakness Vomiting Seizures Papilledema
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Posturing Wide pulse pressure Bradycardia Altered respirations Pupils fixed & dilated
Assessment of IIP Diagnostic tests:
CT scan, MRI Close observation Craig’s screw
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Medical Management of IIP Craniotomy Craniectomy Tumor removal Drainage of ventricles Drainage of hematoma Intubation BGallatin 2007
Medical Management of IIP Medications
Osmotic diuretics - Mannitol Corticosteroids - Decadron Anticonvulsants - Dilantin
Internal monitoring
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Nursing Care of the Patient With IIP Elevate HOB Neck in neutral position Avoid flexion hips, waist and neck Avoid isometric activity or Valsalva
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Restrict fluids Foley Suctioning O2 Hypothermia blanket
Neurological DisordersSeizures Seizures
Disorderly neuron discharges in brain Transitory Different types affect body differently Involuntary movement usually
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Seizures Generalized:
Localized: (Focal)
Tonic-clonic – grand mal Absence - Petit mal Myoclonic Atonic or akinetic
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Partial (Jacksonian) Psychomotor
Seizures Causes:
Hypoglycemia Infection Electrolyte imbalance Trauma IIP Toxins BGallatin 2007
Seizure Medications
Dilantin (Phenytoin) Phenobarbital Mysoline Tridione Valium (Diazepam) Depakene
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Clonopin Mesantoin Neurontin Lamictal Felbatol Cerebyx
Seizure Medications Nursing:
Medications Continue meds Medic alert ID Avoid alcohol, avoid driving, get adequate rest If on Dilantin, instruct on oral hygiene
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Seizures: Nursing Care Protect
Lower to the floor; pad side rails; pillow under head; don’t restrain No bite block or padded tongue blade Allow for post-ictal rest
Prevent aspiration (airway)
Turn side; loosen clothing around neck
Document everything BGallatin 2007
Degenerative Neuro Diseases Multiple Sclerosis
Myasthenia Gravis
Parkinson’s Disease
Amyotrophic Lateral Sclerosis (ALS)
Alzheimer’s Disease Huntington’s Disease (chorea)
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Multiple Sclerosis Common degenerative neurological disease. Myelin sheath is destroyed. Symptoms vary. Relapsing/remitting. Usually ages 20-40.
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Multiple Sclerosis - Symptoms Subjective:
Shakiness, difficulty walking Fatigue, muscle weakness Numbness, tingling Tinnitus Visual problems Difficulty chewing and speaking Incontinent; impotent BGallatin 2007
Multiple Sclerosis - Symptoms Objective:
Ataxia Changes in behavior & emotions Nystagmus Spasticity, tremors, dysphagia, facial palsy, speech impaired, fatigue Incontinence Impaired judgment BGallatin 2007
Multiple Sclerosis - Tests CSF CT scan MRI
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Multiple Sclerosis-Treatment Meds:
Anti inflammatory ACTH,
Solu Medrol, Prednisone
Immuno Modifiers Avonex,
Betaseron, Capoxone
Muscle Relaxants Valium
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Multiple Sclerosis-Nursing Interventions Nutrition Skin Care Activity Control of environment Emotional support Patient teaching BGallatin 2007
Parkinson’s Disease Unknown cause Lack of dopamine. Parkinsonism: encephalitis, toxic chemicals, meds, drugs
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Parkinson’s Symptoms include:
Muscular tremors and rigidity Emotional instability Judgment defects Heat intolerance Mask-like facial appearance Dysphagia and drooling BGallatin 2007
Parkinson’s Testing
No specific test to diagnose Parkinson’s
Diagnosis based on symptoms
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Parkinson’s – Medical Treatment Medications
Sinemet, Symmetrol, Levodopa or Cogentin Less effective over time
Surgery Experimental
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Parkinson’s – Nursing Care Prevent injury (fall or aspiration) Prevent urinary retention and constipation Patient teaching about medication Patient and family support
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Alzheimer’s Unknown cause, but genetic link Very common; risk increases with age Brain changes:
plaques tangled neurons blood vessel degeneration chemical changes BGallatin 2007
Alzheimer’s - Symptoms 1st– memory lapses, difficult word finding, decreased attention span 2nd – increased memory problems, disoriented to time, loses things, confabulates 3rd – total disorientation, apraxia, wanders 4th – severe impairment BGallatin 2007
Alzheimer’s - Testing No definitive test Family history Diagnosis: autopsy
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Alzheimer’s – Medical Management Medication to treat symptoms
Memory:Cognex, Aricept Agitation: Mellaril, Haldol
Supplements
Folic Acid & Vitamin B12 Low fat diet NSAIDS BGallatin 2007
Alzheimer’s – Nursing Care 2 key points for all care:
Prevent overstimulation Provide structured, orderly environment
Other concerns
Communication Family support and education
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Myasthenia Gravis Autoimmune disorder Myoneural junction problem Symptoms:
ptosis, diplopia, weakness, dysarthria, dysphagia, difficulty sitting up, respiratory distress
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Myasthenia Gravis - Treatment Medication
to improve impulse transmission (Mestinon) to suppress immune system (steroids, Cytoxan)
Plasmapheresis Respiratory support Safety
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Amyotrophic Lateral Sclerosis ALS – Lou Gehrig’s disease Motor neurons in brain stem and spinal cord degenerate Brain’s messages don’t reach the muscles Symptoms – weakness, dysarthria, dysphagia No loss of cognitive function No cure, death occurs in 2-6 years
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Huntington’s Disease Chorea Genetic Onset at age 35-45 Excessive involuntary movements Death in 10-20 years No cure
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Huntington’s Disease Nursing interventions are palliative
Give meds Provide for safety Provide adequate diet
Emotional support Genetic counseling
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Cerebrovascular Accident (CVA) Ischemia of brain tissue
Hemorrhage Thrombus Embolus
3rd leading cause of death in the US All ages, but usually elderly BGallatin 2007
CVA – Contributing Factors Atherosclerosis Heart disease Kidney disease
Hypertension Obesity
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High cholesterol Cigarette smoking Stress Sedentary Diabetes Oral contraceptives Cocaine
Cerebral Thrombosis Most common cause of CVA Most often: Atheroclerosis
↓ Thrombus
↓ CVA
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Cerebral Embolism 2nd most common cause of CVA Most often:
Heart disease ↓
Thrombus ↓
Embolus ↓
CVA BGallatin 2007
Cerebral Hemorrhage 3rd most common cause of CVA Most often:
Hypertension ↓
Ruptured cerebral blood vessel ↓
CVA BGallatin 2007
Transient Ischemic Attack Cerebrovascular insufficiency Causes – same as CVA Warning sign of impending CVA
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CVA - Assessment Motor changes
Opposite side Balance, coordination, gait, proprioception Glasgow Coma Scale
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CVA Assessment Sensory Changes
Aphasia =can’t speak or write Agnosia =can’t recognize familiar objects/people Apraxia =can’t perform purposeful acts or use objects properly Neglect Syndrome Visual problems, including hemianopsia BGallatin 2007
CVA Assessment Cognitive changes
denial impaired memory, judgment can’t concentrate disoriented slow and cautious versus impulsive depressed, anxious versus euphoric angers quickly versus constantly smiling BGallatin 2007
CVA - Testing
CT or MRI Cerebral angiogram CBC, PT, PTT, electrolytes
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CVA – Medical Management Thrombolytic (“clot buster”) Anticoagulants Antiplatelet drugs Aneurysm repair Carotid endarterectomy
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CVA-Nursing Care Assess LOC IV, NG, Foley, Vent. Nutrition Encourage perform ADLs Bladder and bowel training ROM Teaching and emotional support BGallatin 2007
Trigeminal Neuralgia Tic Douloureux Trigeminal nerve – degeneration, pressure Facial pain Medication, surgery Avoid triggers
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Bell’s Palsy Facial nerve inflammation Unilateral weakness of facial muscles Steroids, Zovirax, warm moist cloth, massage, facial exercises BGallatin 2007
Infection and Inflammation Meningitis Encephalitis Brain abscess Guillain-Barré
Neurosyphilis Poliomyelitis Herpes zoster AIDS
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Guillain-Barré - Polyneuritis Peripheral nerve disease Prior infection; autoimmune response Weakness and paralysis, begins in extremities and works up Respiratory failure may occur
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Meningitis Acute infection of the meninges Viral or bacterial Severe headache, irritable, fever, delirium, N/V, neck stiffness
Kernig’s sign Brudzinski’s sign BGallatin 2007
Meningitis-Medical Management Diagnosed by LP Medications Respiratory isolation Cool, dark quiet room Maintain hydration Prevent injury BGallatin 2007
Acquired Immunodeficiency Syndrome - AIDS AIDS dementia complex Infection of CNS Dementia Treatment depends on infection Treat symptoms, maintain safety BGallatin 2007
Spinal Cord Trauma Complete cord injury – all voluntary movement below level of trauma is lost Autonomic hyperreflexia
stimulus sympathetic nervous system response
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